Search results for "Beta oxidation"

showing 10 items of 44 documents

Exposure to environmental radionuclides is associated with altered metabolic and immunity pathways in a wild rodent

2019

Wildlife inhabiting environments contaminated by radionuclides face putative detrimental effects of exposure to ionizing radiation, with biomarkers such as an increase in DNA damage and/or oxidative stress commonly associated with radiation exposure. To examine the effects of exposure to radiation on gene expression in wildlife, we conducted a de novo RNA sequencing study of liver and spleen tissues from a rodent, the bank vole Myodes glareolus. Bank voles were collected from the Chernobyl Exclusion Zone (CEZ), where animals were exposed to elevated levels of radionuclides, and from uncontaminated areas near Kyiv, Ukraine. Counter to expectations, we did not observe a strong DNA damage resp…

0106 biological sciences0301 basic medicineRodentDNA Repairmedicine.disease_cause01 natural sciencessäteilybiologiachemistry.chemical_compoundRadiation IonizingMyodes glareolusstable isotopepollutionaineenvaihduntaBeta oxidationradionuclides2. Zero hungerbiologyArvicolinaeFatty AcidsRadiation ExposureRNAseqBank voleMolecular AdaptationLiverimmuunijärjestelmäOriginal ArticleUkraineOxidation-ReductionmetsämyyräDNA damageDNA repair010603 evolutionary biologyMicrobiology03 medical and health sciencesImmunitybiology.animalGeneticsmedicineAnimalsEcology Evolution Behavior and SystematicsRadioisotopesFatty acid metabolismLipid metabolismDNAbiology.organism_classificationLipid MetabolismOxidative Stress030104 developmental biologychemistryChernobyl Nuclear Accident13. Climate actionImmune SystemRNAORIGINAL ARTICLESOxidative stressSpleenDNA DamageMutagens
researchProduct

CD4+ T-cell differentiation and function: Unifying glycolysis, fatty acid oxidation, polyamines NAD mitochondria

2021

The progression through different steps of T-cell development, activation, and effector function is tightly bound to specific cellular metabolic processes. Previous studies established that T-effector cells have a metabolic bias toward aerobic glycolysis, whereas naive and regulatory T cells mainly rely on oxidative phosphorylation. More recently, the field of immunometabolism has drifted away from the notion that mitochondrial metabolism holds little importance in T-cell activation and function. Of note, T cells possess metabolic promiscuity, which allows them to adapt their nutritional requirements according to the tissue environment. Altogether, the integration of these metabolic pathway…

0301 basic medicineChemistryCatabolismImmunologyOxidative phosphorylationMitochondrionCell biologyCitric acid cycle03 medical and health sciencesMetabolic pathway030104 developmental biology0302 clinical medicineAnaerobic glycolysis030220 oncology & carcinogenesisImmunology and AllergyGlycolysisBeta oxidationJournal of Allergy and Clinical Immunology
researchProduct

Circadian and Dopaminergic Regulation of Fatty Acid Oxidation Pathway Genes in Retina and Photoreceptor Cells.

2016

The energy metabolism of the retina might comply with daily changes in energy demand and is impaired in diabetic retinopathy-one of the most common causes of blindness in Europe and the USA. The aim of this study was to investigate putative adaptation of energy metabolism in healthy and diabetic retina. Hence expression analysis of metabolic pathway genes was performed using quantitative polymerase chain reaction, semi-quantitative western blot and immunohistochemistry. Transcriptional profiling of key enzymes of energy metabolism identified transcripts of mitochondrial fatty acid β-oxidation enzymes, i.e. carnitine palmitoyltransferase-1α (Cpt-1α) and medium chain acyl-CoA dehydrogenase (A…

0301 basic medicineMalePhysiologyDopamineMice ObeseGene Expressionlcsh:MedicineBiochemistryAcyl-CoA DehydrogenaseMice0302 clinical medicineCatecholaminesEndocrinologyMedicine and Health SciencesAminesEnzyme Chemistrylcsh:ScienceBeta oxidationMice KnockoutMice Inbred C3HMultidisciplinaryOrganic CompoundsDopaminergicFatty AcidsNeurochemistryDiabetic retinopathyNeurotransmittersCircadian RhythmChemistryCircadian Oscillatorsmedicine.anatomical_structurePhysical SciencesFemaleAnatomyOxidation-Reductionmedicine.drugResearch Articlemedicine.medical_specialtyBiogenic AminesEndocrine DisordersOcular AnatomyBiologyRetinaEnzyme Regulation03 medical and health sciencesOcular SystemInternal medicinemedicineGeneticsDiabetes MellitusAnimalsPhotoreceptor CellsGene RegulationCircadian rhythmCarnitineACADMRetinaDiabetic RetinopathyCarnitine O-PalmitoyltransferaseReceptor Melatonin MT1Receptors Dopamine D4Organic Chemistrylcsh:RChemical CompoundsBiology and Life Sciencesmedicine.diseaseHormonesMice Inbred C57BLMetabolic pathwayDisease Models Animal030104 developmental biologyEndocrinologyMetabolismMicroscopy FluorescenceMetabolic DisordersEnzymologylcsh:Qsense organsEnergy MetabolismPhysiological ProcessesChronobiology030217 neurology & neurosurgeryNeurosciencePLoS ONE
researchProduct

Aerobic growth of Rhodococcus aetherivorans BCP1 using selected naphthenic acids as the sole carbon and energy sources

2018

Naphthenic acids (NAs) are an important group of toxic organic compounds naturally occurring in hydrocarbon deposits. This work shows that Rhodococcus aetherivorans BCP1 cells not only utilize a mixture of eight different NAs (8XNAs) for growth but they are also capable of marked degradation of two model NAs, cyclohexanecarboxylic acid (CHCA) and cyclopentanecarboxylic acid (CPCA) when supplied at concentrations from 50 to 500 mgL−1 . The growth curves of BCP1 on 8XNAs, CHCA, and CPCA showed an initial lag phase not present in growth on glucose, which presumably was related to the toxic effects of NAs on the cell membrane permeability. BCP1 cell adaptation responses that allowed survi…

0301 basic medicineMicrobiology (medical)Inclusion bodie030106 microbiologylcsh:QR1-502Settore BIO/19 - Microbiologia Generale7. Clean energyMicrobiologylcsh:Microbiology03 medical and health scienceschemistry.chemical_compoundBiosynthesisRhodococcus aetherivorans naphthenic acids stress response b-oxidation transmission electron microscopy fatty acids methyl esters inclusion bodiesnaphthenic acidsBeta oxidationchemistry.chemical_classificationbiologyStress responseRhodococcus aetherivoranNaphthenic acidCyclohexanecarboxylic acidbiology.organism_classificationRhodococcus aetherivoranschemistryBiochemistryFatty acids methyl esterβ-oxidationfatty acids methyl estersEnergy sourceRhodococcusBacteriaIntracellularTransmission electron microscopyPolyunsaturated fatty acid
researchProduct

Resveratrol shifts energy metabolism to increase lipid oxidation in healthy old mice.

2019

Abstract Objectives The objective of this work was to determine the specific mechanisms by which resveratrol inhibits lipogenesis and stimulates lipolysis. Methods Twelve male mice were individually introduced into a metabolic cage for 24 h to measure basal metabolic rate, prior to intervention. They were randomly divided into two groups, resveratrol (RSV) and control (C), and administered resveratrol intraperitoneally or vehicle, respectively, for two consecutive days. After 24 h, the metabolic energy expenditure was again determined for 24 h, before mice were sacrificed. Protein and gene expression of different enzymes related to metabolism in the hepatic tissue, adipose tissue and gastro…

0301 basic medicinePolyphenolMalemedicine.medical_specialtyAgingLipolysisAdipose tissueWhite adipose tissueRM1-950ResveratrolLipid catabolism03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicineInternal medicinemedicineAnimalsCarnitineBeta oxidationFatty acid synthesisRespiratory quotientPharmacologyLipogenesisFatty AcidsGeneral MedicineMice Inbred C57BL030104 developmental biologyEndocrinologyMalonyl-CoAchemistryAdipose TissueCarnitine AcyltransferasesLiverResveratrol030220 oncology & carcinogenesisLipogenesisTherapeutics. PharmacologyEnergy MetabolismOxidation-Reductionmedicine.drugAcetyl-CoA CarboxylaseBiomedicinepharmacotherapy = Biomedecinepharmacotherapie
researchProduct

Faecalibacterium prausnitzii treatment improves hepatic health and reduces adipose tissue inflammation in high-fat fed mice

2017

Faecalibacterium prausnitzii is considered as one of the most important bacterial indicators of a healthy gut. We studied the effects of oral F. prausnitzii treatment on high-fat fed mice. Compared to the high-fat control mice, F. prausnitzii-treated mice had lower hepatic fat content, aspartate aminotransferase and alanine aminotransferase, and increased fatty acid oxidation and adiponectin signaling in liver. Hepatic lipidomic analyses revealed decreases in several species of triacylglycerols, phospholipids and cholesteryl esters. Adiponectin expression was increased in the visceral adipose tissue, and the subcutaneous and visceral adipose tissues were more insulin sensitive and less infl…

0301 basic medicinemedicine.medical_specialtyhepatic healthmedicine.medical_treatmentFaecalibacterium prausnitziiAdipose tissueInflammationGut florata3111MicrobiologyMicrobiologyMice03 medical and health sciencesInsulin resistanceInternal medicinemedicineAnimalsIntestinal Mucosaadipose tissue inflammationBeta oxidationEcology Evolution Behavior and SystematicsInflammationgut microbiotaAdiponectinbiologyFaecalibacterium prausnitziiInsulinta1182ta3141Lipid Metabolismbiology.organism_classificationmedicine.diseaseDietary FatsLipids030104 developmental biologyEndocrinologyAdipose TissueLiverOriginal ArticleInsulin Resistancemedicine.symptomThe ISME Journal
researchProduct

Conjugated linoleic acid isomers in mitochondria

2002

The beneficial effects exerted by low amounts of conjugated linoleic acids (C222222237) suggest that CLA are maximally conserved and raise the question about their mitochondrial oxidizability. Cis-9,trans-11-C18:2 (CLA1) and trans-10,cis-12-C18:2 (CLA2) were compared to cis-9,cis-12-C18:2 (linoleic acid; LA) and cis-9-C16:1 (palmitoleic acid; PA), as substrates for total fatty acid (FA) oxidation and for the enzymatic steps required for the entry of FA into rat liver mitochondria. Oxygen consumption rate was lowest when CLA1 was used as a substrate with that on CLA2 being intermediate between it and the respiration on LA and PA. The order of the radiolabeled FA oxidation rate was PA >> LA >…

030309 nutrition & dieteticsConjugated linoleic acidLinoleic acidCAT-IIchemistry.chemical_elementQD415-436BiochemistryOxygenacyl-CoA synthetase03 medical and health scienceschemistry.chemical_compoundEndocrinologymedicinePalmitoleic acidCarnitineBeta oxidation030304 developmental biologychemistry.chemical_classification0303 health sciencescarnitineFatty acidCell BiologyCLAEnzymechemistryBiochemistryCAT-Irespirationmedicine.drugJournal of Lipid Research
researchProduct

AMPK regulates macrophage polarization in adipose tissue inflammation and NASH

2013

Molecular and Translational Medicine, Dept. of Medicine I, University Medical Center, Johannes Gutenberg University Mainz, Langenbeckstrase 1,55116 Mainz, GermanyCOMMENTARY ON:Hematopoietic AMPK beta1 reduces mouse adipose tissue mac-rophage inflammation and insulin resistance in obesity. Galic S,Fullerton MD, Schertzer JD, Sikkema S, Marcinko K, Walkley CR,Izon D, Honeyman J, Chen ZP, van Denderen BJ, Kemp BE, Stein-berg GR. J Clin Invest 2011;121(12):4903–15. Copyright 2011.Reprinted with permission of American Society for ClinicalInvestigation.http://www.ncbi.nlm.nih.gov/pubmed/22080866Abstract: Individuals who are obese are frequently insulin resistant,putting them at increased risk of d…

AMPKmedicine.medical_specialtyMacrophageMacrophage polarizationAdipose tissueAdipose tissueM1M2Insulin resistanceInternal medicineNAFLDmedicineBeta oxidationchemistry.chemical_classificationHepatologybiologyGlucose transporterNASHFatty acidAMPKInsulin resistanceType 2 diabetesmedicine.diseaseFatty acidFibrosisEndocrinologychemistryLiverbiology.proteinGLUT4Research ArticleJournal of Hepatology
researchProduct

Chicken adaptive response to low energy diet: main role of the hypothalamic lipid metabolism revealed by a phenotypic and multi-tissue transcriptomic…

2019

AbstractBackgroundProduction conditions of layer chicken can vary in terms of temperature or diet energy content compared to the controlled environment where pure-bred selection is undertaken. The aim of this study was to better understand the long-term effects of a 15%-energy depleted diet on egg-production, energy homeostasis and metabolism via a multi-tissue transcriptomic analysis. Study was designed to compare effects of the nutritional intervention in two layer chicken lines divergently selected for residual feed intake.ResultsChicken adapted to the diet in terms of production by significantly increasing their feed intake and decreasing their body weight and body fat composition, whil…

Adipose tissueadaptationEnergy homeostasisTranscriptome0302 clinical medicinehypothalamusBeta oxidation2. Zero hunger0303 health sciencesprise alimentaireEndocannabinoid systemAdaptation PhysiologicalCell biologyAlimentation et NutritionBody Composition[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]BiotechnologyResearch Articlelcsh:QH426-470FADS1FADS2lcsh:BiotechnologychickenpouletBiologyModels Biological03 medical and health sciencesQuantitative Trait Heritablelipidlcsh:TP248.13-248.65GeneticsAnimalsFood and Nutritionlipide030304 developmental biologyCaloric RestrictionNeurosciencesLipid metabolismLipid MetabolismDietlcsh:GeneticsGene Expression RegulationNeurons and Cognitionfeed intakeEnergy MetabolismChickenstranscriptome;lipid;feed intake;adaptation;hypothalamus;chickentranscriptome[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgery
researchProduct

Uncoupling protein-2 (UCP2) induces mitochondrial proton leak and increases susceptibility of non-alcoholic steatohepatitis (NASH) liver to ischaemia…

2008

Background: The mechanisms of progression from fatty liver to steatohepatitis and cirrhosis are not well elucidated. Mitochondrial dysfunction represents a key factor in the progression of non-alcoholic steatohepatitis (NASH) as mitochondria are the main cellular site of fatty acid oxidation, ATP synthesis and reactive oxygen species (ROS) production. Aims: (1) To evaluate the role of the uncoupling protein 2 in controlling mitochondrial proton leak and ROS production in NASH rats and humans; and (2) to assess the acute liver damage induced by ischaemia–reperfusion in rats with NASH. Methods: Mitochondria were extracted from the livers of NASH humans and rats fed a methionine and choline de…

AdultMaleMitochondrial ROSmedicine.medical_specialtyMitochondria LiverMitochondrionBiologymedicine.disease_causeIon ChannelsMitochondrial ProteinsAdenosine TriphosphateInternal medicinemedicineAnimalsHumansUncoupling proteinUncoupling Protein 2Rats WistarBeta oxidationAdenosine TriphosphatasesMembrane Potential MitochondrialAldehydesFatty liverGastroenterologyMiddle Agedmedicine.diseaseRatsFatty LiverOxidative StressEndocrinologyMitochondrial respiratory chainLiverBiochemistryReperfusion InjuryAcute DiseaseDisease ProgressionFemaleSteatohepatitisReactive Oxygen SpeciesOxidative stressGut
researchProduct